Search Results

A reduction in brain disorders owing to traumatic brain injury (TBI) caused by head impacts in traffic accidents is needed. However, the details of the injury mechanism still remain unclear. In past analyses, brain parenchyma of a head finite element (FE) model has generally been modeled using simple isotropic viscoelastic materials. For further understanding of TBI mechanism, in this study we developed a new constitutive model that describes most of the mechanical properties in brain parenchyma such as anisotropy, strain rate dependency, and the characteristic features of the unloading process. Validation of the model was performed against several material test data from the literature with a simple one-element model. The model was also introduced into the human head FE model of THUMS v4.02 and validated against post-mortem human subject (PMHS) test data about brain displacements and intracranial pressures during head impacts.

Accurate prediction of occupant head kinematics is critical for better understanding of head/face injury mechanisms in side impacts, especially far-side occupants. In light of the fact that researchers have demonstrated that muscle activations, especially in neck muscles, can affect occupant head kinematics, a human body finite element (FE) model that considers muscle activation is useful for predicting occupant head kinematics in real-world automotive accidents. In this study, we developed a human body FE model called the THUMS (Total HUman Model for Safety) Version 5 that contains 262 one-dimensional (1D) Hill-type muscle models over the entire body. The THUMS was validated against 36 series of PMHS (Post Mortem Human Surrogate) and volunteer test data in this study, and 16 series of PMHS and volunteer test data on side impacts are presented. Validation results with force-time curves were also evaluated quantitatively using the CORA (CORrelation and Analysis) method.

A few reports suggest differences in injury outcomes between cadaver tests and real-world accidents under almost similar conditions. This study hypothesized that muscle activity could primarily cause the differences, and then developed a human body finite element (FE) model with individual muscles. Each muscle was modeled as a hybrid model of bar elements with active properties and solid elements with passive properties. The model without muscle activation was firstly validated against five series of cadaver test data on impact responses in the anterior-posterior direction. The model with muscle activation levels estimated based on electromyography (EMG) data was secondly validated against four series of volunteer test data on bracing effects for stiffness and thickness of an upper arm muscle, and braced driver's responses under a static environment and a brake deceleration.

To investigate the effect of muscle activity in pre-impact on injury outcome, we developed a human arm finite element model with muscles which consisted of solid elements and truss elements that could be used for simulating muscle stiffness change for the inputted activity and 3-D geometry of each muscle. Two series of experimental tests on muscle stiffness change and arm flexion were conducted for validation of the model. Comparisons between the simulation results and test data indicated the model validity. Lateral impact simulations for a left arm demonstrated that the muscle activity in pre-impact had significant effects on the motion and stress distribution of the arm bones.

Injuries of the human brain and spinal cord associated with the central nervous system (CNS) are seen in automotive accidents. CNS injuries are generally categorized into severe injuries (AIS 3+). However, it is not clear how the restraint conditions affect the CNS injuries. This paper presents a newly developed three-dimensional (3D) finite element head-neck model in order to investigate the biomechanical responses of the brain-spinal cord complex. The head model consists of the scalp, skull, and a detailed description of the brain including the cerebrum, cerebellum, brainstem with distinct white and gray matter, cerebral spinal fluid (CSF), sagittal sinus, dura, pia, arachnoid, meninx, falx cerebri, and tentorium. Additionally, the neck model consists of the cervical vertebral bodies, intervertebral discs, muscles, ligaments, spinal cord with white and gray matter, cervical pia, and CSF.