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There has been recent progress over the past 10 years in research comparing 6-year-old thoracic and abdominal response of pediatric volunteers, pediatric post mortem human subjects (PMHS), animal surrogates, and 6-year-old ATDs. Although progress has been made to guide scaling laws of adult to pediatric thorax and abdomen data for use in ATD design and development of finite element models, further effort is needed, particularly with respect to lateral impacts. The objective of the current study was to use the impact response data of age equivalent swine from Yaek et al. (2018) to assess the validity of scaling laws used to develop lateral impact response corridors from adult porcine surrogate equivalents (PSE) to the 3-year-old, 6-year-old, and 10-year-old for the thorax and abdominal body regions.

Analysis and validation of current scaling relationships and existing response corridors using animal surrogate test data is valuable, and may lead to the development of new or improved scaling relationships. For this reason, lateral pendulum impact testing of appropriate size cadaveric porcine surrogates of human 3-year-old, 6-year-old, 10-year-old, and 50th percentile male age equivalence, were performed at the thorax and abdomen body regions to compare swine test data to already established human lateral impact response corridors scaled from the 50th percentile human adult male to the pediatric level to establish viability of current scaling laws. Appropriate Porcine Surrogate Equivalents PSE for the human 3-year-old, 6-year-old, 10-year-old, and 50th percentile male, based on whole body mass, were established. A series of lateral impact thorax and abdomen pendulum testing was performed based on previously established scaled lateral impact assessment test protocols.

An understanding of stiffness characteristics of different body regions, such as thorax, abdomen and pelvis of ES-2re and SID-IIs dummies under controlled laboratory test conditions is essential for development of both compatible performance targets for countermeasures and occupant protection strategies to meet the recently updated FMVSS214, LINCAP and IIHS Dynamic Side Impact Test requirements. The primary purpose of this study is to determine the transfer functions between the ES-2re and SID-IIs dummies for different body regions under identical test conditions using flat rigid wall sled tests. The experimental set-up consists of a flat rigid wall with five instrumented load-wall plates aligned with dummy’s shoulder, thorax, abdomen, pelvis and femur/knee impacting a stationary dummy seated on a rigid low friction seat at a pre-determined velocity.

The main purpose of this study was to determine the impact responses of the different body regions (shoulder, thorax, abdomen and pelvis/leg) of the ES-2re and SID-IIs dummies using rigid wall impacts under different initial test conditions. The experimental set-up consisted of a flat rigid wall with five instrumented load-wall plates aligned with dummy’s shoulder, thorax, abdomen, pelvis and knee impacting a stationary dummy seated on a rigid seat at a pre-determined velocity. The relative location and orientation of the load-wall plates was adjusted relative to the body regions of the ES-2re and SID-IIs dummies respectively.

The main purpose of this study was to determine the impact responses of the different body regions (shoulder, thorax, abdomen and pelvis/leg) of the ES-2re and SID-IIs dummies using rigid wall impacts under different initial test conditions. The experimental set-up consisted of a flat rigid wall with five instrumented load-wall plates aligned with dummy’s shoulder, thorax, abdomen, pelvis and knee impacting a stationary dummy seated on a rigid seat at a pre-determined velocity. The relative location and orientation of the load-wall plates was adjusted relative to the body regions of the ES-2re and SID-IIs dummies respectively.

A modified Marmarou impact acceleration injury model was developed to study the kinematics of the rat head to quantify traumatic axonal injury (TAI) in the corpus callosum (CC) and brainstem pyramidal tract (Py), to determine injury predictors and to establish injury thresholds for severe TAI. Thirty-one anesthetized male Sprague-Dawley rats (392 ± 13 grams) were impacted using a modified impact acceleration injury device from 2.25 m and 1.25 m heights. Beta-amyloid precursor protein (β-APP) immunocytochemistry was used to assess and quantify axonal changes in CC and Py. Over 600 injury maps in CC and Py were constructed in the 31 impacted rats. TAI distribution along the rostro-caudal direction in CC and Py was determined. Linear and angular responses of the rat head were monitored and measured in vivo with an attached accelerometer and angular rate sensor, and were correlated to TAI data.

This study examined the cervical muscle response to physiologic, high-rate (100 mm/s) tensile facet joint capsule (FJC) stretch. Six in-vivo caprine C5/6 FJC preparations were subjected to an incremental tensile loading paradigm. EMG activity was recorded from the right trapezius (TR) and multifidus (MF) muscle groups at the C5 and C6 levels; and from the sternomastoid (SM) and longus colli (LC) muscle groups bilaterally at the C5/6 level; during FJC stretch. Capsule load during the displacement applications was recorded via a miniature load cell, and 3D capsule strains (based on stereoimaging of an array of markers on the capsule surface) were reconstructed using finite element methods. EMG traces from each muscle were examined for onset of muscular activity. Capsule strains and loads at the time of EMG onset were recorded for each muscle, as was the time from the onset of FJC stretch to the onset of muscle activity. All muscles were responsive to physiologic high-rate FJC stretch.

Traumatic brain injury (TBI) continues to be a major health problem, with over 500,000 cases per year with a societal cost of approximately $85 billion in the US. Motor vehicle accidents are the leading cause of such injuries. In many cases of TBI widespread disruption of the axons occurs through a process known as diffuse axonal injury (DAI) or traumatic axonal injury (TAI). In the current study, an in vivo TAI model was developed using spinal nerve roots of adult rats. This model was used to determine functional and structural responses of axons to various strains and displacement rates. Fifty-six L5 dorsal nerve roots were each subjected to a predetermined strain range (<10%, 10-20% and >20%) at a specified displacement rate (0.01 mm/sec and 15 mm/sec) only once. Image analysis was used to determine actual strains on the roots during the pull.

In the automotive seating industry measurements of lumbar support prominence and height are performed to assess their effects on occupant comfort. This project investigated measurement methods for lumbar support prominence and height in an occupied seat. Fifteen participants provided subjective responses of their perceived lumbar support prominence and height utilizing specifically developed visual analog scales. Also, pressure measurements were taken while the participants were seated. The recently developed H-point manikin II was utilized as a standardized sitter. Specifically, the lumbar support prominence (LSP) measure was used for the prominence measures. Pressure mat readings with the seated manikin was used for lumbar support height determination and prominence correlations. With both manikin and participants in the seat, the lumbar support was digitized through the rear of the seat.

Cervical facet joints are implicated as a major source of pain after whiplash injury. The purpose of this study was to investigate the proposed capsule strain injury mechanism of whiplash pain using neurophysiologic methods. Strain thresholds, threshold distribution, saturation strains and afterdischarge responses of capsule neural receptors were characterized in vivo. Goat C5-C6 facet joint capsules were used to identify and characterize capsule receptors in response to controlled uniaxial stretch by recording C6 dorsal rootlet nerve discharge. The joints were stretched at 0.5 mm/sec in a series of tests with 2 mm increments until the capsule ruptured. Ninety-two identified units were responsive to physiologic or noxious stretch while 28 were silent receptors. Among the 50 characterized responsive units, 42 showed low strain thresholds at 10.2±4.6% while 8 had high strain thresholds at 47.2±9.6%.

To date, several lateral impact studies (Bolte et al., 2000, 2003, Marth, 2002 and Compigne et al., 2004) have been performed on the shoulder to determine the response characteristics and injury threshold of the shoulder complex. Our understanding of the biomechanical response and injury tolerance of the shoulder would be improved if the results of these tests were combined. From a larger data base shoulder injury tolerance criteria can be developed as well as corridors for side impact dummies. Data from the study by Marth (2002, 12 tests) was combined with data from the previous studies. Twenty-two low speed tests (4.5 ± 0.7 m/s) and 9 high speed tests (6.7 ± 0.7 m/s) were selected from the combined data for developing corridors. Shoulder force, deflection and T1y acceleration corridors were developed using a minimization of cumulative variance technique.

Traumatic rupture of the aorta (TRA) is a leading cause of death in high velocity blunt trauma, particularly motor vehicle accidents. However, little is understood about the mechanisms of TRA and thus, the opportunities to prevent TRA in the motor vehicle environment are compromised. The objective of this study was to determine the relationship between impact response and TRA through analyses of data from cadaver tests that successfully produced TRA in lateral impacts. Seventeen Heidelberg-style side impact sled tests were conducted using unembalmed human cadavers. Three sled speeds were used: 6.7, 9.0, and 10.5 m/s. Three barrier configurations were used: rigid flat wall, rigid wall with a 152-mm offset toward the pelvis, and a flat wall with padding of varying stiffness. Multiple load and acceleration measurements were made on the barrier and cadaver. Potential injury parameters were evaluated and their relative predictive abilities were examined.

Thirty-three fresh human cadaver shoulders were harvested and bone-ligament-bone specimens of acromioclavicular joint, coracoclavicular joint and sternoclavicular joint were obtained. A test fixture and clamps specifically designed for this ligament study and a high-speed Instron machine were used. One quasi-static rate (nominally 0.1 %/sec) and two high rates (nominally, high rate 1 = 40,000 %/sec and high rate 2 = 15,000 %/sec) were used in this study. In the acromioclavicular joint tests, ligament failure was the most common failure mode. Bone fractures occurred most often at the clavicle rather than acromion. In the coracoclavicular joint tests, the majority of specimens failed at the ligament and bone fractures occurred at the coracoid. In the sternoclavicular joint tests, the specimen failed at the bone in most cases. In the acromioclavicular joint and coracoclavicular joint tests, high rate 2 tests and quasi-static tests had more bone fracture cases than high rate 1 tests.

The biomechanical response and injury tolerance of the shoulder in lateral impacts is not well understood. These data are needed to better understand human injury tolerance, validate finite element models and develop biofidelic shoulders in side impact dummies. Seventeen side impact sled tests were performed with unembalmed human cadavers. Data analyzed for this study include T1-Y acceleration, shoulder and thoracic load plate forces, upper sternum x and y accelerations, and struck side acromion x, y and z accelerations. One dimensional deflection at the shoulder level was determined from high-speed film by measuring the distance between a target on T1 and the impacted wall. Force-time response corridors were obtained for tests with 9 m/s pelvic offset, 10.5 m/s pelvic offset, 9 m/s unpadded flat wall, 6.7 m/s unpadded flat wall, 9 m/s soft padding and 9 m/s stiff padding. Maximum shoulder plate forces in unpadded 9 m/s tests (5.5 kN) were larger than in 6.7 m/s tests (3.3 kN).

The purpose of this study is to help understand the thoracic response and injury mechanisms in high-energy, limited-stroke, lateral velocity pulse impacts to the human chest wall. To impart such impacts, a linear impactor was developed which had a limited stroke and minimally decreased velocity during impact. The peak impact velocity was 5.6 ± 0.3 m/s. A series of BioSID and cadaver tests were conducted to measure biomechanical response and injury data. The conflicting effects of padding on increased deflection and decreased acceleration were demonstrated in tests with BioSID and cadavers. The results of tests conducted on six cadavers were used to test several proposed injury criteria for side impact. Linear regression was used to correlate each injury criterion to the number of rib fractures. This test methodology captured and supported a contrasting trend of increased chest deflection and decreased TTI when padding was introduced.

A sled-to-sled subsystem side impact test methodology has been developed by using two sleds at the WSU Bioengineering Center in order to simulate a car-to-car side impact, particularly in regards to the door velocity profile. Initially this study concentrated on tailoring door pulse to match the inner door velocity profile from FMVSS 214 full-scale dynamic side impact tests. This test device simulates a pulse quite similar to a typical door velocity of a full size car in a dynamic side impact test. Using the newly developed side impact test device three runs with a SID dummy were performed to study the effects of door padding and spacing in a real side impact situation. This paper describes the test methodology to simulate door intrusion velocity profiles in side impact and discusses SID dummy test results for different padding conditions.

The purpose of this paper is to address abdominal injury and response in cadaver whole body side impacts and abdominal injury risk functions in SID and BIOSID in whole body impacts. Side impact sled tests were performed at Wayne State University using cadavers, SID and BIOSID, with response measured at the shoulder, thorax, abdominal and pelvic levels. The data at the abdominal level are presented here. These data provide further understanding of abdominal tolerance and response in lateral impact and the ability of side impact dummies to predict abdominal injury. In addition, the padding data provide insight into tolerable armrest loads.

Thirty-seven SID side impact sled tests were performed using a rigid wall and a padded wall with fourteen different padding configurations. The Thoracic Trauma Index (TTI) and Average Spine Acceleration (ASA) were measured in each test. TTI and ASA were evaluated in terms of their ability to predict injury in identical cadaver tests and in terms of their ability to predict the harm or benefit of padding of different crush strengths. SID ASA predicted the injury seen in WSU-CDC cadaver tests better than SID TTI. SID ASA predicted that padding of greater than 20 psi crush strength is harmful (ASA > 40 g's). SID TTI predicted that padding of greater than 20 psi crush strength is beneficial (TTI < 85 g's). SID TTI predicts the benefit of lower impact velocity. However, SID ASA appears more useful in assessing the harm or benefit of door padding or air bags.

Seventeen Heidelberg type cadaveric side impact sled tests, two sled-to-sled tests, and forty-four pendulum tests have been conducted at Wayne State University, to determine human responses and tolerances in lateral collisions. This paper describes the development of a simplified finite element model of a human occupant in a side impact configuration to simulate those cadaveric experiments. The twelve ribs were modeled by shell elements. The visceral contents were modeled as an elastic solid accompanied by an array of discrete dampers. Bone condition factors were obtained after autopsy to provide material properties for the model. The major parameters used for comparison are contact forces at the level of shoulder, thorax, abdomen and pelvis, lateral accelerations of ribs 4 and 8 and of T12, thoracic compression and injury functions V*C, TTI and ASA.

Three-dimensional film analysis was used to study the response of the shoulder and thoracic skeleton of cadavers to lateral sled tests conducted at Wayne State University. The response of the shoulder structure was of particular interest, although, it is perhaps the most difficult skeletal structure to track in a side impact. Results of the three-dimensional film analysis are given for rigid impacts at 6.7 and 9.1 meters per second, and for padded impacts averaging 9 meters per second. Results from a two-dimensional film analysis are included for the impacted clavicle which could not be tracked by the three-dimensional film analysis. Displacements at various locations on the shoulder and thoracic skeleton were normalized to estimate the response of a fiftieth percentile male.