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Experimentally derived brain response envelopes are needed to evaluate and validate existing finite element (FE) head models. Motion of the brain relative to the skull during rotational input was measured using high-speed biplane x-ray. To generate repeatable, reproducible, and scalable data, methods were developed to reduce experimental variance. An “extreme-energy” device was developed to provide a controlled input that is unaffected by specimen characteristics. Additionally, a stereotactic frame was used to deploy radiopaque markers at specific, pre-determined locations within the brain. One post-mortem human surrogate (PMHS) head specimen was subjected to repeat tests of a half-sine rotational speed pulse in the sagittal plane. The desired pulse had a peak angular speed of 40 rad/s and duration of 30 ms. Relative motion of the brain was quantified using radiopaque targets and high-speed biplane x-ray. Frontal and occipital intracranial pressure (ICP) were also measured.

High-speed biplane x-ray was used to investigate relative kinematics of the thoracoabdominal organs in response to blunt loading. Four post-mortem human surrogates instrumented with radiopaque markers were subjected to eight crash-specific loading scenarios, including frontal chest and abdominal impacts, as well as driver-shoulder seatbelt loading. Testing was conducted with each surrogate perfused, ventilated, and positioned in an inverted, fixed-back configuration. Displacement of radiopaque markers recorded with high-speed x-ray in two perspectives was tracked using motion analysis software and projected into calibrated three-dimensional coordinates. Internal organ kinematics in response to blunt impact were quantified for the pericardium, lungs, diaphragm, liver, spleen, stomach, mesentery, and bony structures.

A total of eight low-speed, rear-end impact tests using two Post Mortem Human Subjects (PMHS) in a seated posture are reported. These tests were conducted using a HYGE-style mini-sled. Two test conditions were employed: 8 kph without a headrestraint or 16 kph with a headrestraint. Upper-body kinematics were captured for each test using a combination of transducers and high-speed video. A 3-2-2-2-accelerometer package was used to measure the generalized 3D kinematics of both the head and pelvis. An angular rate sensor and two single-axis linear accelerometers were used to measure angular speed, angular acceleration, and linear acceleration of T1 in the sagittal plane. Two high-speed video cameras were used to track targets rigidly attached to the head, T1, and pelvis. The cervical spine kinematics were captured with a high-speed, biplane x-ray system by tracking radiopaque markers implanted into each cervical vertebra.

The objectives of this study were to examine the response, repeatability, and injury predictive ability of the Hybrid III small-female dummy to static out-of-position (OOP) deployments using a depowered driver-side airbag. Five dummy tests were conducted in two OOP configurations by two different laboratories. The OOP configurations were nose-on-rim (NOR) and chest-on-bag (COB). Four cadaver tests were conducted using unembalmed small-female cadavers and the same airbags used in the dummy tests under similar OOP conditions. One cadaver test was designed to increase airbag loading of the face and neck (a forehead-on-rim, or FOR test). Comparison between the dummy tests of Lab 1 and of Lab 2 indicated the test conditions and results were repeatable. In the cadaver tests no skull fractures or neck injuries occurred. However, all four cadavers had multiple rib fractures.

This study investigated the mechanisms of traumatic rupture of the aorta (TRA). Eight unembalmed human cadavers were tested using various dynamic blunt loading modes. Impacts were conducted using a 32-kg impactor with a 152-mm face, and high-speed seatbelt pretensioners. High-speed biplane x-ray was used to visualize aortic motion within the mediastinum, and to measure deformation of the aorta. An axillary thoracotomy approach was used to access the peri-isthmic region to place radiopaque markers on the aorta. The cadavers were inverted for testing. Clinically relevant TRA was observed in seven of the tests. Peak average longitudinal Lagrange strain was 0.644, with the average peak for all tests being 0.208 ± 0.216. Peak intraluminal pressure of 165 kPa was recorded. Longitudinal stretch of the aorta was found to be a principal component of injury causation. Stretch of the aorta was generated by thoracic deformation, which is required for injury to occur.

High-speed biplane x-ray and neutral density targets were used to examine brain displacement and deformation during impact. Relative motion, maximum principal strain, maximum shear strain, and intracranial pressure were measured in thirty-five impacts using eight human cadaver head and neck specimens. The effect of a helmet was evaluated. During impact, local brain tissue tends to keep its position and shape with respect to the inertial frame, resulting in relative motion between the brain and skull and deformation of the brain. The local brain motions tend to follow looping patterns. Similar patterns are observed for impact in different planes, with some degree of posterior-anterior and right-left symmetry. Peak coup pressure and pressure rate increase with increasing linear acceleration, but coup pressure pulse duration decreases. Peak average maximum principal strain and maximum shear are on the order of 0.09 for CFC 60 Hz data for these tests.

This study focuses on the biaxial mechanical properties of planar aorta tissue at strain rates likely to be experienced during automotive crashes. It also examines the structural response of the whole aorta to longitudinal tension. Twenty-six tissue-level tests were conducted using twelve thoracic aortas harvested from human cadavers. Cruciate samples were excised from the ascending, peri-isthmic, and descending regions. The samples were subjected to equibiaxial stretch at two nominal speed levels using a new biaxial tissue-testing device. Inertia-compensated loads were measured to facilitate calculation of true stress. High-speed videography and regional correlation analysis were used to track ink dots marked on the center of each sample to obtain strain. In a series of component-level tests, the response of the intact thoracic aorta to longitudinal stretch was obtained using seven aorta specimens. The aorta fails within the peri-isthmic region.

Traumatic rupture of the aorta (TRA) is an important transportation-related injury. This study investigated TRA mechanisms using in situ human cadaver experiments. Four quasi-static tests and one dynamic test were performed. The quasi-static experiments were conducted by perturbing the mediastinal structures of the cadavers. The mechanisms investigated included anterior, superior, and lateral displacement of the heart and aortic arch. The resulting injuries ranged from partial tears to complete transections. All injuries occurred within the peri-isthmic region. Intimal tears were associated with the primary injuries. The average failure load and stretch were 148 N and 30 percent for the quasi-static tests. This study illustrates that TRA can result from appropriate application of nominal levels of longitudinal load and tension. The results demonstrate that intraluminal pressure and whole-body acceleration are not required for TRA to occur.

This study characterizes the response of the human cadaver abdomen to high-speed seatbelt loading using pyrotechnic pretensioners. A test apparatus was developed to deliver symmetric loading to the abdomen using a seatbelt equipped with two low-mass load cells. Eight subjects were tested under worst-case scenario, out-of-position (OOP) conditions. A seatbelt was placed at the level of mid-umbilicus and drawn back along the sides of the specimens, which were seated upright using a fixed-back configuration. Penetration was measured by a laser, which tracked the anterior aspect of the abdomen, and by high-speed video. Additionally, aortic pressure was monitored. Three different pretensioner designs were used, referred to as system A, system B and system C. The B and C systems employed single pretensioners. The A system consisted of two B system pretensioners. The vascular systems of the subjects were perfused.

A biaxial test device was designed to obtain the material properties of aortic tissue at rates consistent with those seen in automotive impact. Fundamental to the design are four small tissue clamps used to grasp the ends of the tissue sample. The applied load at each clamp is determined using subminiature load cells in conjunction with miniature accelerometers for inertial compensation. Four lightweight carriages serve as mounting points for each clamp. The carriages ride on linear shafts, and are equipped with low-friction bearings. Each carriage is connected to the top of a central drive disk by a rigid link. A fifth carriage, also connected to the drive disk by a rigid link, is attached at the bottom. A pneumatic cylinder attached to the lower carriage initiates rotation of the disk. This produces identical motion of the upper carriages in four directions away from the disk center.

One of the leading causes of death in automotive crashes is traumatic rupture of the aorta (TRA) or blunt aortic injury (BAI). The risk of fatality is high if an aortic injury is not detected and treated promptly. The objective of this study is to investigate TRA mechanisms using finite element (FE) simulations of reconstructed real-world accidents involving aortic injury. For this application, a case was obtained from the William Lehman Injury Research Center (WLIRC), which is a Crash Injury Research and Engineering Network (CIREN) center. In this selected crash, the case vehicle was struck on the left side with a Principal Direction of Force (PDoF) of 290 degrees. The side structure of the case vehicle crushed a maximum of 0.33 m. The total delta-V was estimated to be 6.2 m/s. The occupant, a 62-year old mid-sized male, was fatally injured. The occupant sustained multiple rib fractures, laceration of the right ventricle, and TRA, among other injuries.

Traumatic rupture of the aorta (TRA) is a leading cause of death in high velocity blunt trauma, particularly motor vehicle accidents. However, little is understood about the mechanisms of TRA and thus, the opportunities to prevent TRA in the motor vehicle environment are compromised. The objective of this study was to determine the relationship between impact response and TRA through analyses of data from cadaver tests that successfully produced TRA in lateral impacts. Seventeen Heidelberg-style side impact sled tests were conducted using unembalmed human cadavers. Three sled speeds were used: 6.7, 9.0, and 10.5 m/s. Three barrier configurations were used: rigid flat wall, rigid wall with a 152-mm offset toward the pelvis, and a flat wall with padding of varying stiffness. Multiple load and acceleration measurements were made on the barrier and cadaver. Potential injury parameters were evaluated and their relative predictive abilities were examined.

Knee injuries represent about 10% of all injuries suffered during car crashes. Efforts to assess the injury risk to the posterior cruciate ligament (PCL) have been based on a study available in the literature (Viano et al., 1978), in which only two of the five knees tested had PCL ruptures. The aims of the current study were to repeat the study with a higher number of samples, study the effects of other soft tissues on knee response, and assess the adequacy of the experimental setup for the identification of a PCL tolerance. A total of 14 knees were tested using a high-speed materials testing machine. Eight were intact knees (with the patella and all the muscular and ligamentous structures), three were PCL-only knees (patella and all the muscular and ligamentous structures other than the PCL removed), and the last three were PCL-only knees with the tibia protected from bending fracture.

The two main motivations for Wayne State University (WSU) and Henry Ford Hospital (HFH) researchers to develop numerical human surrogates are advanced computing technology and a high-speed x-ray imaging device not available just a decade ago. This paper summarizes the capabilities and limitations of detailed component models of the human body, from head to foot, developed at WSU over the last decade (Zhang et al. 2001, Yang et al. 1998, Shah et al. 2001, Iwamoto et al. 2000, Lee et al. 2001 and Beillas et al. 2001). All of these models were validated against global response data obtained from relevant high-speed cadaveric tests. Additionally, some models were also validated against local kinematics of bones or soft tissues obtained using the high-speed x-ray system. All of these models have been scaled to conform to the key dimensions of a 50th percentile male.

A parameterized, or scalable, finite element (FE) model of the human head was developed and validated against the available cadaver experiment data for three impact directions (frontal, occipital and lateral). The brain material properties were modeled using a hyperelastic and viscoelastic constitutive law. The interface between the skull and the brain was modeled in three different ways ranging from purely tied (no-slip) to sliding (free-slip). Two sliding contact definitions were compared with the tied condition. Also, three different stiffness parameters, encompassing the range of published brain tissue properties, were tested. The model using the tied contact definition correlated well with the experimental results for the coup and contrecoup pressures in a frontal impact while the sliding interface models did not. Relative motion between the skull and the brain in low-severity impacts appears to be relatively insensitive to the contact definitions.

Brain tissue architecture consists of a complex network of neurons and vasculature interspersed within a matrix of supporting cells. The role of the relatively suffer blood vessels on the more compliant brain tissues during rapid loading has not been properly investigated. Two 2-D finite element models of the human head were developed. The basic model (Model I) consisted of the skull, dura matter, cerebral spinal fluid (CSF), tentorium, brain tissue and the parasagittal bridging veins. The pia mater was also included but in a simplified form which does not correspond to the convolutions of the brain. In Model II, major branches of the cerebral arteries were added to Model I. Material properties for the brain tissues and vasculature were taken from those reported in the literature. The model was first validated against intracranial pressure and brain/skull relative motion data from cadaveric tests.

Aortic injuries during blunt thoracic impacts can lead to life threatening hemorrhagic shock and potential exsanguination. Experimental approaches designed to study the mechanism of aortic rupture such as the testing of cadavers is not only expensive and time consuming, but has also been relatively unsuccessful. The objective of this study was to develop a computer model and to use it to predict modes of loading that are most likely to produce aortic ruptures. Previously, a 3D finite element model of the human thorax was developed and validated against data obtained from lateral pendulum tests. The model included a detailed description of the heart, lungs, rib cage, sternum, spine, diaphragm, major blood vessels and intercostal muscles. However, the aorta was modeled as a hollow tube using shell elements with no fluid within, and its material properties were assumed to be linear and isotropic.

This study was conducted to resolve discrepancies and fill in gaps in the biomechanical impact response of the human abdomen to frontal impact loading. Three types of abdominal loading were studied: rigid-bar impacts, seatbelt loading, and close-proximity (out-of-position) airbag deployments. Eleven rigid-bar free-back tests were performed into the mid and upper abdomens of unembalmed instrumented human cadavers using nominal impact speeds of 6 and 9 m/s. Seven fixed-back rigid-bar tests were also conducted at 3, 6, and 9 m/s using one cadaver to examine the effects of body mass, spinal flexion, and repeated testing. Load-penetration corridors were developed and compared to those previously established by other researchers. Six seatbelt tests were conducted using three cadavers and a peak-loading rate of 3 m/s. The seatbelt loading tests were designed to maximize belt/abdomen interaction and were not necessarily representative of real-world crashes.

The objective of this work was to develop a reusable, rate-sensitive dummy abdomen with abdominal injury assessment capability. The primary goal for the abdomen developed was to have good biofidelity in a variety of loading situations that might be encountered in an automotive collision. This paper presents a review of previous designs for crash dummy abdomens, a description of the development of the new abdomen, results of testing with the new abdomen and instrumentation, and suggestions for future work. The biomechanical response targets for the new abdomen were determined from tests of the mid abdomen done in a companion biomechanical study. The response of the abdominal insert is an aggregate response of the dummy’s entire abdominal area and does not address differences in upper versus lower abdominal response, solid versus hollow organs, or organ position or mobility.

This study continued the biomechanical investigations of forearm fractures caused by direct loading of steering-wheel airbags during the early stages of deployment. Twenty-four static deployments of driver airbags were conducted into the forearms of unembalmed whole cadavers using a range of airbags, including airbags that are depowered as allowed by the new federal requirements for frontal impact testing. In general, the depowered airbags showed a reduction in incidence and severity of forearm fractures compared to the pre-depowered airbags tested. Data from these twenty-four tests were combined with results from previous studies to develop a refined empirical model for fracture occurrence based on Average Distal Forearm Speed (ADFS), and a revised value for fifty-percent probability of forearm-bone fracture of 10.5 m/s. Bone mineral content, which is directly related to forearm tolerance, was found to be linearly related to arm mass.